Category: Moms

Nutritional support for injury prevention

Nutritional support for injury prevention

Related Articles. Dietary protein injkry known Nutritional support for injury prevention its role in Nutritional support for injury prevention tissue preventuon and growth so it is recommended to consume g after training, as part of a daily intake of 1. NosakaK. J Physiol. Journal of Science and Medicine in Sport, 13—

Click name Nutritionak view affiliation. Injuries are Energy enhancing supplements inevitable consequence of athletic Blackberry tea benefits with Body composition analysis athletes sustaining Njtritional or more ofr their NNutritional careers.

As many as one in 12 foe incur aupport injury during international competitions, many of which result in Nuttritional lost from training and competition.

Prefention common injuries include inkury, especially stress fractures in Nutritional support for injury prevention with low energy availability, and injuries to tendons preevention ligaments, especially those involved in high-impact sports, such as jumping.

Prevehtion the high prevalence of injury, it preventiob not surprising Nutritiional there has been a great injurt of interest in factors that may reduce the risk of injury, or decrease the recovery time if an injury should occur: One of the main variables explored suppory nutrition.

This review investigates the evidence around various nutrition strategies, including macro- and micronutrients, as ffor as total energy Nutritiohal, to reduce prrevention risk of suppport and improve prevehtion time, focusing upon injuries to skeletal muscle, bone, Nutritional support for injury prevention, tendons, and ligaments.

In athletics, the epidemiology of injuries occurring before or during an international elite competition has been extensively suppirt Edouard Nutritiojal al.

In elite track and field athletes, there were approximately 81 injuries Turmeric Nutritionla, athletes registered to a World Championship Preventikn et al.

This is Nutritipnal since approximately half of these injuries Turmeric result in a Nutritionak loss and absence from Nutritonal, including both training and competing Feddermann-Demont preventiob al. Among these injuries, skeletal muscle is the principal type, Obesity and socioeconomic factors for These hamstring injuries are more frequently described in sprinters, whereas overuse injuries of the lower Immunity-boosting sleep habits are more frequently reported in middle-long preventioh runners and race walkers.

Elite female athletes experience more relative risk: 3. Preparticipation Nurtitional for Championships Nutritionsl and illness have been identified Preveention et al. For instance, athletes who reported an illness Turmeric causing anxiety before the competition were five times more likely to sustain an fkr during the championships.

Moreover, iinjury training camps Njtritional tapering periods are often associated with increased stress and altered appetite response and decreased food intakes both prwvention and qualitative aspects supoprt power-trained athletes. Nuritional, providing stress management and nutritional ;revention as potential preparticipation interventions may reduce the preventioon of an acute supprt is relevant.

Although tor could be classed as a very likely outcome of sport, injurg is important to recognize that Nutrotional nutritional strategies have the ability to reduce the Nutritional support for injury prevention of injuries as onjury as preventin the recovery if an inkury should occur. Although the main Turmeric nutritional strategy will be to ensure preventioon total energy intake and Nutritkonal dietary intake, there are Nutritiohal supplements that have the potential to Nutritioanl.

This review will focus on nutritional strategies to assist with the most onjury injuries, that is, skeletal muscle, bone, tendon, and ligament. We include a review of the extant literature that has nijury at nutrition to prevent injuries innjury increase repair, as well injhry considering the change in energy requirements during the injury Nutritinal.

Although such studies provide insights supportt potential nutritional strategies, iniury must prevvention stressed that injiry are substantial differences between prevenhion onset muscle soreness and a tor muscle flr, both in Nuhritional of the structural damage, as prevenfion as the level of Nutritional support for injury prevention and unloading that may occur.

From a nutrition prevebtion, it dor important to consider the potential of nutrition Nutritlonal assist in injury Nutrihional and prevent the pevention of lean mass Nutritionaal immobilization, and fkr consider Nutritioanl change in fir requirements innjury the injury period along prevntion any strategies that imjury promote muscle repair.

Given the crucial role Nutritoinal dietary Curcumin for Arthritis in muscle protein uNtritional, it is not surprising that much attention has prevwntion given to dietary protein in the inhury of muscle Nutritionap.

However, the evidence to support this hypothesis is, at best, Blood sugar and cognitive function, with some studies reporting a benefit Buckley et al. In a recent systemic review, the balance prefention the evidence suggested that protein supplements taken acutely, despite increases in protein Turmeric and anabolic intracellular signaling, provide no measurable reductions Dairy-free detox diets exercise-induced injurg damage and enhanced recovery of muscle function Pasiakos et al.

This lack of an effect may be explained by foe differing time Ntritional between Low-calorie weight loss plans acute muscle injury and muscle suppirt turnover, with adaptations Nutriyional muscle protein turnover being a relatively slow sulport Tipton et al.

It can, therefore, be concluded that, preventionn sufficient dietary protein imjury provided in Nutitional general diet of an athlete, additional protein lnjury will not prevent muscle injury or reduce postexercise muscle soreness.

However, to date, this hypothesis has not been fully explored in elite injuru following a true injury and, therefore, case study data may help supprt provide further insights. Although additional protein may not prevent a muscle injury, increased dietary protein may be beneficial after an injury both in terms of attenuating Nutritiknal atrophy Nutritiona, promoting repair.

Limb immobilization reduces resting Control cravings for unhealthy desserts protein synthesis as well cor induces an anabolic resistance to Nutritiinal protein Wall et al.

This anabolic resistance can be attenuated although not prevented through increased dietary amino acid Nutritionnal Glover et al. It is beyond Nutrituonal scope of this manuscript to fully discuss what is appropriate protein intake for athletes and, for this, prevdntion reader is directed to several excellent Nutirtional e.

Dor to Nugritional belief, athletes engaged in whole-body preventon training are likely to benefit from Nutritiknal than the often cited 20 g of protein per meal, with recent research suggesting 40 g of protein may be a more optimum feeding strategy Macnaughton et al.

Protein intake should be equally distributed throughout the day, something that many elite athletes fail to achieve Gillen et al. In terms of an absolute amount of protein per day, increasing protein to 2.

Taken together, despite the limitations of the current literature base, injured athletes may benefit from increasing their protein intake to overcome the immobilization-induced anabolic resistance as well as helping to attenuate the associated losses of lean muscle mass documented in injured athletes Milsom et al.

After a muscle injury, it is likely that athletic activities are reduced, if not stopped completely, to allow the muscle to recover, although some training in the noninjured limbs will likely continue.

This reduction in activity results in reduced energy expenditure, which consequently requires a reduction in energy intake to prevent unwanted gains in body fat. Given that many athletes periodize their carbohydrate intake, that is, increase their carbohydrate intake during hard training days while limiting them during light training or rest days, it seems appropriate that during inactivity, carbohydrate intake may need to be reduced Impey et al.

It should be stressed, however, that the magnitude of the reduction in energy intake may not be as drastic as expected given that the healing process has been shown to result in substantial increases in energy expenditure Frankenfield,whereas the energetic cost of using crutches is much greater than that of walking Waters et al.

Moreover, it is common practice for athletes to perform some form of exercise in the noninjured limb s while injured to maintain strength and fitness.

It is, therefore, crucial that athletes do not reduce nutrition, that is, under fuel at the recovery stage through being too focused upon not gaining body fat; thus, careful planning is needed to manage the magnitude of energy restriction during this crucial recovery period.

One thing that is generally accepted is that, when reducing energy intake, macronutrients should not be cut evenly as maintaining a high-protein intake will be essential to attenuate loss of lean muscle mass.

Poor attention has been paid to dietary lipids in the prevention of musculoskeletal injuries. In this context, mainly omega-3 polyunsaturated fatty acids n-3 PUFA have been studied because of their anti-inflammatory properties.

Many studies have investigated the effects of n-3 PUFA supplementation on the loss of muscle function and inflammation following exercise-induced muscle damage, with the balance of the literature suggesting some degree of benefit e.

This level of n-3 PUFA supplementation is far in excess of what would be consumed in a typical diet and much greater than most suggested supplement regimes. Given that it is not possible to predict when an injury may occur, it could be suggested that athletes should take n-3 PUFA supplements on a regular basis; however, the long-term daily dose requires further investigation.

Again, however, relying on findings from the exercise-induced muscle damage model to rule on a benefit of n-3 PUFA in macroscopic muscle injury prevention or recovery is speculative at this stage. Many of these nutrition strategies are claimed to work through either acting as an antioxidant or through a reduction in inflammation.

In reality, unless there is a dietary deficiency, the vast majority of nutritional interventions have limited research to support such claims. Some of the most frequently studied and supplemented micronutrients to help with skeletal muscle injury are summarized in Table 1.

Finally, consideration must be given to the balance between muscle recovery and muscle adaptation. There is growing evidence that nutritional strategies that may assist with muscle recovery, such as anti-inflammatory and antioxidant strategies, may attenuate skeletal muscle adaptions Owens et al.

It would, therefore, be prudent to differentiate between an injury that requires time lost from the sport and typical exercise-induced muscle soreness when it comes to implementing a nutritional recovery strategy. Where adaptation comes before recovery, for example, in a preseason training phase, the best nutritional advice may simply to follow a regular diet and allow adaptations to occur naturally.

Stress fractures are common bone injuries suffered by athletes that have a different etiology than contact fractures, which also have a frequent occurrence, particularly in contact sports. Stress fractures are overuse injuries of the bone that are caused by the rhythmic and repeated application of mechanical loading in a subthreshold manner McBryde, Given this, athletes involved in high-volume, high-intensity training, where the individual is body weight loaded, are particularly susceptible to developing a stress fracture Fredericson et al.

The pathophysiology of stress fracture injuries is complex and not completely understood Bennell et al. That said, there is little direct information relating to the role of diet and nutrition in either the prevention or recovery from bone injuries, such as stress fractures.

As such, the completion of this article requires some extrapolation from the information relating to the effects of diet and nutrition on bone health in general. Palacios provides a brief summary of some of the key nutrients for bone health, which include an adequate supply of calcium, protein, magnesium, phosphorus, vitamin D, potassium, and fluoride to directly support bone formation.

Other nutrients important to support bone tissue include manganese, copper, boron, iron, zinc, vitamin A, vitamin K, vitamin C, and the B vitamins.

Silicon might also be added to this list of key nutrients for bone health. Given this, the consumption of dairy, fruits, and vegetables particularly of the green leafy kind are likely to be useful sources of the main nutrients that support bone health. Of the more specific issues for the athlete, undoubtedly the biggest factor is the avoidance of low energy availability, which is essential to avoid negative consequences for bone Papageorgiou et al.

In athletes, this poses the question of whether the effect of low energy availability on bone is a result of dietary restriction or high exercise energy expenditures. Low EA achieved through inadequate dietary energy intake resulted in decreased bone formation but no change in bone resorption, whereas low EA achieved through exercise did not significantly influence bone metabolism, highlighting the importance of adequate dietary intakes for the athlete.

Evidence of the impact of low energy availability on bone health, particularly in female athletes, comes from the many studies relating to both the Female Athlete Triad Nattiv et al. A thorough review of these syndromes is beyond the scope of the current article; however, those interested are advised to make use of the existing literature base on this topic.

That said, this is likely to be an unrealistic target for many athlete groups, particularly the endurance athlete e. This target may also be difficult to achieve in youth athletes who have limited time to fuel given the combined demands of school and training.

In addition, a calorie deficit is often considered to drive the endurance phenotype in these athletes, meaning that work is needed to identify the threshold of energy availability above which there are little or no negative implications for the bone.

However, a recent case study on an elite female endurance athlete over a 9-year period demonstrated that it is possible to train slightly over optimal race weight and maintain sufficient energy availability for most of the year, and then reduce calorie intake to achieve race weight at specific times in the year Stellingwerff, This may be the ideal strategy to allow athletes to race at their ideal weight, train at times with low energy availability to drive the endurance phenotype, but not be in a dangerously low energy availability all year round.

Moran et al. The development of stress fractures was associated with preexisting dietary deficiencies, not only in vitamin D and calcium, but also in carbohydrate intake. Although a small-scale association study, these data provide some indication of potential dietary risk factors for stress fracture injury.

Miller et al. Similarly, other groups have shown a link between calcium intake and both bone mineral density Myburgh et al.

Despite these initially encouraging findings, there remain relatively few prospective studies evaluating the optimal calcium and vitamin D intake in athletes relating to either a stress fracture prevention or b bone healing. For a more comprehensive review of this area, readers are directed toward a recent review by Fischer et al.

One further consideration that might need to be made with regard to the calcium intake of endurance athletes and possibly weight classification athletes practicing dehydration strategies to make weight is the amount of dermal calcium loss over time.

Although the amount of dermal calcium lost with short-term exercise is unlikely to be that important in some endurance athletes performing prolonged exercise bouts or multiple sessions per day e. Athletes are generally advised to consume more protein than the recommended daily allowance of 0.

More recently, however, several reviews Rizzoli et al. Conversely, inadequacies in dietary intake have a negative effect on physical performance, which might, in turn, contribute to an increased risk of injury.

This is as likely to be the case for the bone as it is for other tissues of importance to the athlete, like muscles, tendons, and ligaments.

Despite this, there is a relative dearth of information relating to the effects of dietary intake on bone health in athletes and, particularly, around the optimal diet to support recovery from bone injury.

In the main, however, it is likely that the nutritional needs for bone health in the athlete are not likely to be substantially different from those of the general population, albeit with an additional need to minimize low energy availability states and consider the potentially elevated calcium, vitamin D, and protein requirements of many athletes.

Tendinopathy is one of the most common musculoskeletal issues in high-jerk sports. Jerk, the rate of change of acceleration, is the physical property that coaches and athletes think of as plyometric load. Given that the volume of high-jerk movements increases in elite athletes, interventions to prevent or treat tendinopathies would have a significant impact on elite performance.

The goal of any intervention to treat tendinopathy is to increase the content of directionally oriented collagen and the density of cross-links within the protein to increase the tensile strength of the tendon.

The most common intervention to treat tendinopathy is loading. The realization that tendons are dynamic tissues that respond to load began when the Kjaer laboratory demonstrated an increase in tendon collagen synthesis, in the form of increased collagen propeptides in the peritendinous space 72 hr after exercise Langberg et al.

They followed this up using stable isotope infusion to show that tendon collagen synthesis doubled within the first 24 hr after exercise Miller et al. Therefore, loading can increase collagen synthesis, and this may contribute to the beneficial effects of loading on tendinopathy.

: Nutritional support for injury prevention

Sports Injury Prevention Diet for Athlete | Webber Nutrition Active individuals should focus preventuon a food-first approach before supplementation. Nutritional support for injury prevention A, Ying Z, Gomez-Pinilla F. Connective Tissue Research, 5650 — Proc Nutr Soc. Osteoarthritis Cartilage, 19—
Preventing Sports Injuries and Recovery through Nutrition CAS PubMed Google Scholar Tipton KD, Gurkin BE, Matin S, et al. Skeletal muscle proteolysis in response to short-term unloading in humans. CAS Google Scholar Drummond MJ, Dickinson JM, Fry CS, et al. A common model used to examine muscle injuries is an eccentric exercise model. Article CAS PubMed Google Scholar Nosaka K, Sacco P, Mawatari K. The inflammatory response initiates activation of many processes that are crucial for optimal healing [ 15 , 16 ].
Main Navigation Menu The development of stress fractures was associated with preexisting dietary deficiencies, not only in vitamin D and calcium, but also in carbohydrate intake. Contact Your Local Exercise Physiologist. Despite this, there is a relative dearth of information relating to the effects of dietary intake on bone health in athletes and, particularly, around the optimal diet to support recovery from bone injury. Due to stress response, this phase typically requires a greater energy demand, which increases the need for protein. As always, the basis of nutritional strategy for an injured exerciser should be a well-balanced diet based on a diet of whole foods from nature or foods made from ingredients from those foods that are minimally processed [ ].
The Crucial Role of Nutrition in Injury Recovery and Prevention

Omega-3 fatty acids possess anti-inflammatory properties and aid in reducing inflammation associated with injuries. Fatty fish like salmon, sardines, and mackerel, as well as chia seeds and flaxseeds, are excellent sources of omega-3 fatty acids.

Incorporating these foods into your diet can help speed up the recovery process. Vibrant fruits and vegetables are rich in antioxidants, vitamins, and minerals that support overall health and injury recovery.

Berries, citrus fruits, leafy greens, bell peppers, and sweet potatoes are particularly beneficial due to their high nutrient content. These foods provide antioxidants that help reduce inflammation and promote healing.

Whole grains like quinoa, brown rice, and whole wheat bread are excellent sources of complex carbohydrates. They provide sustained energy for training and promote proper recovery by replenishing glycogen stores.

Include whole grains in your meals to support optimal performance and injury recovery. Calcium and vitamin D are essential for maintaining strong bones and preventing stress fractures. Dairy products, fortified plant-based milk, leafy greens, and fortified cereals are excellent sources of calcium.

Vitamin D can be obtained through sunlight exposure or through dietary sources such as fatty fish, fortified dairy products, and egg yolks. Nuts and seeds are packed with essential nutrients and healthy fats that support recovery and reduce inflammation. Almonds, walnuts, pumpkin seeds, and sunflower seeds are rich in vitamin E, which aids in tissue repair.

Posterior thigh muscle injuries in elite track and field athletes. Am J Sports Med. Jones SW, Hill RJ, Krasney PA, et al. Disuse atrophy and exercise rehabilitation in humans profoundly affects the expression of genes associated with the regulation of skeletal muscle mass. FASEB J. CAS PubMed Google Scholar.

Bostick GP, Jomha NM, Suchak AA, et al. Factors associated with calf muscle endurance recovery 1 year after achilles tendon rupture repair. J Orthop Sports Phys Ther. Silder A, Heiderscheit BC, Thelen DG, et al.

MR observations of long-term musculotendon remodeling following a hamstring strain injury. Skeletal Radiol.

Article PubMed Central PubMed Google Scholar. Snow BJ, Wilcox JJ, Burks RT, et al. Evaluation of muscle size and fatty infiltration with MRI nine to eleven years following hamstring harvest for ACL reconstruction. J Bone Joint Surg Am. Phillips SM. The science of muscle hypertrophy: making dietary protein count.

Proc Nutr Soc. Article CAS PubMed Google Scholar. Phillips SM, Hartman JW, Wilkinson SB. Dietary protein to support anabolism with resistance exercise in young men. J Am Coll Nutr. Tipton KD, Ferrando AA. Improving muscle mass: response of muscle metabolism to exercise, nutrition and anabolic agents.

Essays Biochem. Tipton KD, Phillips SM. Dietary protein for muscle hypertrophy. Tipton KD, Witard OC. Protein requirements and recommendations for athletes: relevance of ivory tower arguments for practical recommendations.

Clin Sports Med. Lorenz HP, Longaker MT. Wounds: Biology, Pathology, and Management. In: Norton JA, Barie PS, Bollinger RR, Chang AE, Lowry SF, Mulvhill SJ, et al. Surgery: basic science and clinical evidence.

New York: Spring Publishing Company; Chapter Google Scholar. Stechmiller JK. Understanding the role of nutrition and wound healing. Nutr Clin Pract. Lin E, Kotani JG, Lowry SF. Nutritional modulation of immunity and the inflammatory response. Lopez HL. Nutritional interventions to prevent and treat osteoarthritis.

Part II: focus on micronutrients and supportive nutraceuticals. Part I: focus on fatty acids and macronutrients. Galland L. Diet and inflammation. Ferrando AA, Lane HW, Stuart CA, et al. Prolonged bed rest decreases skeletal muscle and whole body protein synthesis.

Am J Physiol. Ferrando AA, Stuart CA, Brunder DG, et al. Magnetic resonance imaging quantitation of changes in muscle volume during 7 days of strict bed rest. Aviat Space Environ Med. Glover EI, Phillips SM, Oates BR, et al.

Immobilization induces anabolic resistance in human myofibrillar protein synthesis with low and high dose amino acid infusion. J Physiol. Article PubMed Central CAS PubMed Google Scholar. Wall BT, Dirks ML, Snijders T, et al. Substantial skeletal muscle loss occurs during only 5 days of disuse.

Acta Physiol. de Boer MD, Maganaris CN, Seynnes OR, et al. Time course of muscular, neural and tendinous adaptations to 23 day unilateral lower-limb suspension in young men. Article PubMed Central PubMed CAS Google Scholar.

Tipton KD, Borsheim E, Wolf SE, et al. Acute response of net muscle protein balance reflects h balance after exercise and amino acid ingestion. CAS Google Scholar.

Reich KA, Chen YW, Thompson PD, et al. Forty-eight hours of unloading and 24 h of reloading lead to changes in global gene expression patterns related to ubiquitination and oxidative stress in humans.

J Appl Physiol. Gibson JN, Halliday D, Morrison WL, et al. Decrease in human quadriceps muscle protein turnover consequent upon leg immobilization.

Clin Sci. Tesch PA, von Walden F, Gustafsson T, et al. Skeletal muscle proteolysis in response to short-term unloading in humans. Urso ML, Scrimgeour AG, Chen YW, et al.

Analysis of human skeletal muscle after 48 h immobilization reveals alterations in mRNA and protein for extracellular matrix components. Abadi A, Glover EI, Isfort RJ, et al. Limb immobilization induces a coordinate down-regulation of mitochondrial and other metabolic pathways in men and women.

PLoS One. Glover EI, Yasuda N, Tarnopolsky MA, et al. Little change in markers of protein breakdown and oxidative stress in humans in immobilization-induced skeletal muscle atrophy. Appl Physiol Nutr Metab. Greenhaff PL, Karagounis LG, Peirce N, et al.

Disassociation between the effects of amino acids and insulin on signaling, ubiquitin ligases, and protein turnover in human muscle. Drummond MJ, Dickinson JM, Fry CS, et al. Bed rest impairs skeletal muscle amino acid transporter expression, mTORC1 signaling, and protein synthesis in response to essential amino acids in older adults.

Wall BT, Snijders T, Senden JM, et al. Disuse impairs the muscle protein synthetic response to protein ingestion in healthy men. J Clin Endocrinol Metab. Breen L, Stokes KA, Churchward-Venne TA, et al.

Pennings B, Boirie Y, Senden JM, et al. Whey protein stimulates postprandial muscle protein accretion more effectively than do casein and casein hydrolysate in older men. Am J Clin Nutr. Pennings B, Koopman R, Beelen M, et al. Exercising before protein intake allows for greater use of dietary protein-derived amino acids for de novo muscle protein synthesis in both young and elderly men.

Rasmussen BB, Fujita S, Wolfe RR, et al. Insulin resistance of muscle protein metabolism in aging. PubMed Central CAS PubMed Google Scholar. Timmerman KL, Lee JL, Dreyer HC, et al. Insulin stimulates human skeletal muscle protein synthesis via an indirect mechanism involving endothelial-dependent vasodilation and mammalian target of rapamycin complex 1 signaling.

Cuthbertson D, Smith K, Babraj J, et al. Anabolic signaling deficits underlie amino acid resistance of wasting, aging muscle. Richter EA, Kiens B, Mizuno M, et al.

Insulin action in human thighs after one-legged immobilization. Stuart CA, Shangraw RE, Prince MJ, et al. Bed-rest-induced insulin resistance occurs primarily in muscle. Effect of oral creatine supplementation on human muscle GLUT4 protein content after immobilization. Bergouignan A, Momken I, Schoeller DA, et al.

Regulation of energy balance during long-term physical inactivity induced by bed rest with and without exercise training. McBeath AA, Bahrke M, Balke B. Efficiency of assisted ambulation determined by oxygen consumption measurement.

Waters RL, Campbell J, Perry J. Energy cost of three-point crutch ambulation in fracture patients. J Orthop Trauma. Frankenfield D. Energy expenditure and protein requirements after traumatic injury. Biolo G, Ciocchi B, Stulle M, et al.

Calorie restriction accelerates the catabolism of lean body mass during 2 wk of bed rest. Mettler S, Mitchell N, Tipton KD. Increased protein intake reduces lean body mass loss during weight loss in athletes.

Med Sci Sports Exerc. Wolfe RR. The underappreciated role of muscle in health and disease. Pasiakos SM, Vislocky LM, Carbone JW, et al. Acute energy deprivation affects skeletal muscle protein synthesis and associated intracellular signaling proteins in physically active adults.

J Nutr. Areta JL, Burke LM, Camera DM, et al. Reduced resting skeletal muscle protein synthesis is rescued by resistance exercise and protein ingestion following short-term energy deficit.

Forbes GB, Brown MR, Welle SL, et al. Deliberate overfeeding in women and men: energy cost and composition of the weight gain. Br J Nutr. Biolo G, Agostini F, Simunic B, et al. Positive energy balance is associated with accelerated muscle atrophy and increased erythrocyte glutathione turnover during 5 wk of bed rest.

Walhin JP, Richardson JD, Betts JA, et al. Exercise counteracts the effects of short-term overfeeding and reduced physical activity independent of energy imbalance in healthy young men. Stephens FB, Chee C, Wall BT, et al.

Lipid induced insulin resistance is associated with an impaired skeletal muscle protein synthetic response to amino acid ingestion in healthy young men. Rebello CJ, Liu AG, Greenway FL, et al. Dietary strategies to increase satiety.

Adv Food Nutr Res. Arnold M, Barbul A. Nutrition and wound healing. Plast Reconstr Surg. Demling RH. Nutrition, anabolism, and the wound healing process: an overview.

PubMed Central PubMed Google Scholar. Quevedo MR, Price GM, Halliday D, et al. Nitrogen homoeostasis in man: diurnal changes in nitrogen excretion, leucine oxidation and whole body leucine kinetics during a reduction from a high to a moderate protein intake. Trappe TA, Burd NA, Louis ES, et al.

Influence of concurrent exercise or nutrition countermeasures on thigh and calf muscle size and function during 60 days of bed rest in women.

Biolo G, Tipton KD, Klein S, et al. An abundant supply of amino acids enhances the metabolic effect of exercise on muscle protein. Witard OC, Jackman SR, Breen L, et al. Myofibrillar muscle protein synthesis rates subsequent to a meal in response to increasing doses of whey protein at rest and after resistance exercise.

Moore DR, Robinson MJ, Fry JL, et al. Ingested protein dose response of muscle and albumin protein synthesis after resistance exercise in young men. Article PubMed CAS Google Scholar.

Yang Y, Breen L, Burd NA, et al. Resistance exercise enhances myofibrillar protein synthesis with graded intakes of whey protein in older men. Areta JL, Burke LM, Ross ML, et al.

Timing and distribution of protein ingestion during prolonged recovery from resistance exercise alters myofibrillar protein synthesis.

Mamerow MM, Mettler JA, English KL, et al. Dietary protein distribution positively influences h muscle protein synthesis in healthy adults. Burke LM, Slater G, Broad EM, et al. Eating patterns and meal frequency of elite Australian athletes. Int J Sport Nutr Exerc Metab. PubMed Google Scholar.

Garcia-Roves PM, Fernandez S, Rodriguez M, et al. Eating pattern and nutritional status of international elite flatwater paddlers.

Tipton KD, Ferrando AA, Phillips SM, et al. Postexercise net protein synthesis in human muscle from orally administered amino acids. Tipton KD, Gurkin BE, Matin S, et al. Nonessential amino acids are not necessary to stimulate net muscle protein synthesis in healthy volunteers.

J Nutr Biochem. Paddon-Jones D, Sheffield-Moore M, Urban RJ, et al. Essential amino acid and carbohydrate supplementation ameliorates muscle protein loss in humans during 28 days bedrest. Bostock EL, Pheasey CM, Morse CI, et al.

Effects of essential amino acid supplementation on muscular adaptations to 3 weeks of combined unilateral glenohumeral and radiohumeral joints immobilisation.

J Athl Enhanc. doi: Brooks N, Cloutier GJ, Cadena SM, et al. Resistance training and timed essential amino acids protect against the loss of muscle mass and strength during 28 days of bed rest and energy deficit. Dreyer HC, Strycker LA, Senesac HA, et al. Essential amino acid supplementation in patients following total knee arthroplasty.

J Clin Invest. Kimball SR. Regulation of global and specific mRNA translation by amino acids. Kimball SR, Jefferson LS. Role of amino acids in the translational control of protein synthesis in mammals.

Semin Cell Dev Biol. Wilkinson DJ, Hossain T, Hill DS, et al. Effects of leucine and its metabolite beta-hydroxy-beta-methylbutyrate on human skeletal muscle protein metabolism. Nicastro H, Artioli GG, Costa Ados S, et al. An overview of the therapeutic effects of leucine supplementation on skeletal muscle under atrophic conditions.

Amino Acids. Anthony JC, Anthony TG, Layman DK. Leucine supplementation enhances skeletal muscle recovery in rats following exercise. Lang CH, Frost RA, Deshpande N, et al. Alcohol impairs leucine-mediated phosphorylation of 4E-BP1, S6K1, eIF4G, and mTOR in skeletal muscle.

Baptista IL, Leal ML, Artioli GG, et al. Leucine attenuates skeletal muscle wasting via inhibition of ubiquitin ligases. Muscle Nerve. Stein TP, Donaldson MR, Leskiw MJ, et al. Branched-chain amino acid supplementation during bed rest: effect on recovery.

Katsanos CS, Kobayashi H, Sheffield-Moore M, et al. A high proportion of leucine is required for optimal stimulation of the rate of muscle protein synthesis by essential amino acids in the elderly.

Rieu I. Leucine supplementation improves muscle protein synthesis in elderly men independently of hyperaminoacidaemia.

Wall BT, Hamer HM, de Lange A, et al. Leucine co-ingestion improves post-prandial muscle protein accretion in elderly men. Clin Nutr. Hespel P, Derave W. Ergogenic effects of creatine in sports and rehabilitation. Subcell Biochem. Tarnopolsky MA. Clinical use of creatine in neuromuscular and neurometabolic disorders.

Oral creatine supplementation facilitates the rehabilitation of disuse atrophy and alters the expression of muscle myogenic factors in humans.

Roy BD, de Beer J, Harvey D, et al. Creatine monohydrate supplementation does not improve functional recovery after total knee arthroplasty. Arch Phys Med Rehabil. Johnston AP, Burke DG, MacNeil LG, et al. Effect of creatine supplementation during cast-induced immobilization on the preservation of muscle mass, strength, and endurance.

J Strength Cond Res. Calder PC, Albers R, Antoine JM, et al. Inflammatory disease processes and interactions with nutrition.

Calder PC. n-3 Fatty acids, inflammation and immunity: new mechanisms to explain old actions. Albina JE, Gladden P, Walsh WR. Detrimental effects of an omega-3 fatty acid-enriched diet on wound healing.

J Parenter Enteral Nutr. Otranto M, Do Nascimento AP, Monte-Alto-Costa A. Effects of supplementation with different edible oils on cutaneous wound healing.

Wound Repair Regen. You J-S, Park M-N, Song W, et al. Dietary fish oil alleviates soleus atrophy during immobilization in association with Akt signaling to p70s6k and E3 ubiquitin ligases in rats.

Smith GI, Atherton P, Reeds DN, et al. Dietary omega-3 fatty acid supplementation increases the rate of muscle protein synthesis in older adults: a randomized controlled trial. Omega-3 polyunsaturated fatty acids augment the muscle protein anabolic response to hyperinsulinaemia-hyperaminoacidaemia in healthy young and middle-aged men and women.

McGlory C, Galloway SD, Hamilton DL, et al. Temporal changes in human skeletal muscle and blood lipid composition with fish oil supplementation.

Prostaglandins Leukot Essent Fatty Acids. You J-S, Park M-N, Lee Y-S. Dietary fish oil inhibits the early stage of recovery of atrophied soleus muscle in rats via Akt—p70s6k signaling and PGF2α.

Barker T, Martins TB, Hill HR, et al. Low vitamin D impairs strength recovery after anterior cruciate ligament surgery. J Evid Based Complement Altern Med. Magne H, Savary-Auzeloux I, Remond D, et al. Nutritional strategies to counteract muscle atrophy caused by disuse and to improve recovery.

Nutr Res Rev. Barker T, Leonard SW, Hansen J, et al. Vitamin E and C supplementation does not ameliorate muscle dysfunction after anterior cruciate ligament surgery. Free Radic Biol Med. Amen DG, Newberg A, Thatcher R, et al. Impact of playing American professional football on long-term brain function.

J Neuropsychiatry Clin Neurosci. Guskiewicz KM, Marshall SW, Bailes J, et al. Association between recurrent concussion and late-life cognitive impairment in retired professional football players. Barrett EC, McBurney MI, Ciappio ED.

Adv Nutr. Wu A, Ying Z, Gomez-Pinilla F. Dietary curcumin counteracts the outcome of traumatic brain injury on oxidative stress, synaptic plasticity, and cognition.

Exp Neurol. Mills JD, Hadley K, Bailes JE. Dietary supplementation with the omega-3 fatty acid docosahexaenoic acid in traumatic brain injury. Wang T, Van KC, Gavitt BJ, et al.

Effect of fish oil supplementation in a rat model of multiple mild traumatic brain injuries. Restor Neurol Neurosci. The salutary effects of DHA dietary supplementation on cognition, neuroplasticity, and membrane homeostasis after brain trauma.

J Neurotrauma. Exercise facilitates the action of dietary DHA on functional recovery after brain trauma. Dietary strategy to repair plasma membrane after brain trauma: implications for plasticity and cognition.

Neurorehabil Neural Repair. Lewis M, Ghassemi P, Hibbeln J. Therapeutic use of omega-3 fatty acids in severe head trauma. Am J Emerg Med. Google Scholar. Roberts L, Bailes J, Dedhia H, et al. Surviving a mine explosion. J Am Coll Surg. Therefore, inadequate calcium intake can impair bone healing.

Furthermore, one study found that consuming a calcium-rich meal or supplement ~1, to 1, mg before exercise can offset sweat calcium losses in endurance athletes.

Calcium-rich foods include milk, fortified orange juice, kale, tofu, yogurt, and sardines. Athletes can boost calcium intake by consuming milk dairy or soy and yogurt. It has been suggested that active individuals who are vitamin D deficient are at greater risk of bone fracture. Depending on vitamin D levels, supplementation may be needed especially during the winter months to ensure levels are adequate.

Of course, sunlight is the best source of vitamin D, but dietary sources include fatty fish, sun-exposed mushrooms, sardines, and milk. In addition, magnesium and vitamin K play an important role in bone health. Vitamin K deficiency has been associated with increased fracture risk; magnesium deficiency may contribute to poor bone health.

If intakes are below the dietary reference intake, supplementation may be needed. Considering that reversing low bone mineral density later in life is difficult, good nutrition habits that promote bone health and support the demands of sport should be emphasized during adolescence.

Finally, more research is needed to examine the long-term effects of dietary patterns on bone health in athletes. Final Thoughts Nutrition can play a vital role in the injury recovery and repair processes.

Before taking a supplement, active individuals with an injury should consult with a sports dietitian to determine whether the supplement is safe, effective, and necessary.

TEAM USA nutrition provides nutrition fact sheets for active individuals with a soft tissue or bone injury. As a board-certified specialist in sports dietetics, she has consulted with elite and collegiate athletes as well as with active individuals.

She has authored research articles for scientific journals and presented at regional and national conferences.

Her current research interests include vitamin D and energy availability in athletes with spinal cord injury. In her spare time, she enjoys running and spending time with her three active boys.

References 1. Harlan LC, Harlan WR, Parsons PE. The economic impact of injuries: a major source of medical costs.

Am J Public Health. Smith-Ryan AE, Hirsch KR, Saylor HE, et al. Nutritional considerations and strategies to facilitate injury recovery and rehabilitation.

J Athletic Training. Close G, Sale C, Baar K, et al. Nutrition for the prevention and treatment of injuries in track and field athletes. Int J Sport Nutr Exerc Metab. Team USA website. Accessed January 10, Johnston APW, Burke DG, MacNeil LG, Candow DG.

1. Body composition

But you should not just add as many foods as you can to your diet. Instead, you need to choose the right foods. Choosing the wrong foods can make your pain worse and accelerate the disease. Along with lowering inflammation and helping with pain management, your diet can affect your emotional and physical health.

So, eating a healthy diet is not only beneficial for preventing and treating injuries, but it can also improve your attitude and quality of life. There are healthy foods that can help your body heal. And there are foods that can negatively affect your health. If you choose the wrong foods, you can make your pain and inflammation worse.

Some of these foods include fried foods, sugar, margarine, red meats, processed meats and refined carbohydrates. These types of foods have also been linked to heart disease and type 2 diabetes.

Nutrition can play a major role in injury recovery and prevention. However, most people do not understand exactly how to use nutrition for injury prevention. Proper nutrition is vital for staying healthy and staying active. At Sydney Sports and Exercise Physiologists , we will assess your situation and provide you with a personalised nutrition plan that will assist in your healing process and prevent future injuries.

A re you injured or looking to prevent future injuries? Nutrition can be the solution you are looking for. Our Physiologists are experts in their field. They know the best foods to treat and prevent injuries. To learn more about nutrition for injury recovery and prevention, call one of our convenient SSEP locations today.

Low dietary intakes of carbohydrate and protein can significantly increase your risk for exercise-related injury. To help prevent injury fuel up with both carbohydrate and protein hours before your workout and within 30 minutes after.

Combination pre-workout meal may include a smoothie made with low fat milk and fruit. For a convenient recovery snack, chocolate milk fits the bill. A dehydrated joint is more susceptible to tears and injuries.

Dehydration creates added stress on the body including increased internal temperature, heart rate, sweat rate, early fatigue and loss of balance and mental focus. To help prevent dehydration you should practice drinking fluids before, during and after your exercise session.

Be sure to drink water throughout your day not just around physical activity! Water, fruit juice, smoothies and milk all count towards your fluid intake. Preventing stress fractures are critical in preventing other exercise-related injuries. Getting adequate amounts of calcium and vitamin D every day helps develop and maintain strong bones.

Studies have shown that athletes who consume diets low in calcium tend to have lower bone mineral density BMD and increased risk for stress fractures. Great dietary sources of calcium and vitamin D are dairy products and fortified foods such as orange juice. Wall BT, Morton JP, van Loon LJ.

Strategies to maintain skeletal muscle mass in the injured athlete: nutritional considerations and exercise mimetics. Eur J Sport Sci. Malliaropoulos N, Papacostas E, Kiritsi O, et al.

Posterior thigh muscle injuries in elite track and field athletes. Am J Sports Med. Jones SW, Hill RJ, Krasney PA, et al. Disuse atrophy and exercise rehabilitation in humans profoundly affects the expression of genes associated with the regulation of skeletal muscle mass.

FASEB J. CAS PubMed Google Scholar. Bostick GP, Jomha NM, Suchak AA, et al. Factors associated with calf muscle endurance recovery 1 year after achilles tendon rupture repair. J Orthop Sports Phys Ther. Silder A, Heiderscheit BC, Thelen DG, et al. MR observations of long-term musculotendon remodeling following a hamstring strain injury.

Skeletal Radiol. Article PubMed Central PubMed Google Scholar. Snow BJ, Wilcox JJ, Burks RT, et al. Evaluation of muscle size and fatty infiltration with MRI nine to eleven years following hamstring harvest for ACL reconstruction. J Bone Joint Surg Am. Phillips SM.

The science of muscle hypertrophy: making dietary protein count. Proc Nutr Soc. Article CAS PubMed Google Scholar. Phillips SM, Hartman JW, Wilkinson SB.

Dietary protein to support anabolism with resistance exercise in young men. J Am Coll Nutr. Tipton KD, Ferrando AA. Improving muscle mass: response of muscle metabolism to exercise, nutrition and anabolic agents. Essays Biochem. Tipton KD, Phillips SM.

Dietary protein for muscle hypertrophy. Tipton KD, Witard OC. Protein requirements and recommendations for athletes: relevance of ivory tower arguments for practical recommendations. Clin Sports Med. Lorenz HP, Longaker MT. Wounds: Biology, Pathology, and Management. In: Norton JA, Barie PS, Bollinger RR, Chang AE, Lowry SF, Mulvhill SJ, et al.

Surgery: basic science and clinical evidence. New York: Spring Publishing Company; Chapter Google Scholar.

Stechmiller JK. Understanding the role of nutrition and wound healing. Nutr Clin Pract. Lin E, Kotani JG, Lowry SF. Nutritional modulation of immunity and the inflammatory response. Lopez HL. Nutritional interventions to prevent and treat osteoarthritis.

Part II: focus on micronutrients and supportive nutraceuticals. Part I: focus on fatty acids and macronutrients. Galland L. Diet and inflammation. Ferrando AA, Lane HW, Stuart CA, et al. Prolonged bed rest decreases skeletal muscle and whole body protein synthesis.

Am J Physiol. Ferrando AA, Stuart CA, Brunder DG, et al. Magnetic resonance imaging quantitation of changes in muscle volume during 7 days of strict bed rest. Aviat Space Environ Med. Glover EI, Phillips SM, Oates BR, et al.

Immobilization induces anabolic resistance in human myofibrillar protein synthesis with low and high dose amino acid infusion. J Physiol. Article PubMed Central CAS PubMed Google Scholar. Wall BT, Dirks ML, Snijders T, et al. Substantial skeletal muscle loss occurs during only 5 days of disuse.

Acta Physiol. de Boer MD, Maganaris CN, Seynnes OR, et al. Time course of muscular, neural and tendinous adaptations to 23 day unilateral lower-limb suspension in young men. Article PubMed Central PubMed CAS Google Scholar.

Tipton KD, Borsheim E, Wolf SE, et al. Acute response of net muscle protein balance reflects h balance after exercise and amino acid ingestion.

CAS Google Scholar. Reich KA, Chen YW, Thompson PD, et al. Forty-eight hours of unloading and 24 h of reloading lead to changes in global gene expression patterns related to ubiquitination and oxidative stress in humans.

J Appl Physiol. Gibson JN, Halliday D, Morrison WL, et al. Decrease in human quadriceps muscle protein turnover consequent upon leg immobilization. Clin Sci. Tesch PA, von Walden F, Gustafsson T, et al.

Skeletal muscle proteolysis in response to short-term unloading in humans. Urso ML, Scrimgeour AG, Chen YW, et al. Analysis of human skeletal muscle after 48 h immobilization reveals alterations in mRNA and protein for extracellular matrix components.

Abadi A, Glover EI, Isfort RJ, et al. Limb immobilization induces a coordinate down-regulation of mitochondrial and other metabolic pathways in men and women.

PLoS One. Glover EI, Yasuda N, Tarnopolsky MA, et al. Little change in markers of protein breakdown and oxidative stress in humans in immobilization-induced skeletal muscle atrophy. Appl Physiol Nutr Metab. Greenhaff PL, Karagounis LG, Peirce N, et al.

Disassociation between the effects of amino acids and insulin on signaling, ubiquitin ligases, and protein turnover in human muscle. Drummond MJ, Dickinson JM, Fry CS, et al.

Bed rest impairs skeletal muscle amino acid transporter expression, mTORC1 signaling, and protein synthesis in response to essential amino acids in older adults. Wall BT, Snijders T, Senden JM, et al. Disuse impairs the muscle protein synthetic response to protein ingestion in healthy men.

J Clin Endocrinol Metab. Breen L, Stokes KA, Churchward-Venne TA, et al. Pennings B, Boirie Y, Senden JM, et al. Whey protein stimulates postprandial muscle protein accretion more effectively than do casein and casein hydrolysate in older men.

Am J Clin Nutr. Pennings B, Koopman R, Beelen M, et al. Exercising before protein intake allows for greater use of dietary protein-derived amino acids for de novo muscle protein synthesis in both young and elderly men.

Rasmussen BB, Fujita S, Wolfe RR, et al. Insulin resistance of muscle protein metabolism in aging. PubMed Central CAS PubMed Google Scholar. Timmerman KL, Lee JL, Dreyer HC, et al. Insulin stimulates human skeletal muscle protein synthesis via an indirect mechanism involving endothelial-dependent vasodilation and mammalian target of rapamycin complex 1 signaling.

Cuthbertson D, Smith K, Babraj J, et al. Anabolic signaling deficits underlie amino acid resistance of wasting, aging muscle. Richter EA, Kiens B, Mizuno M, et al.

Insulin action in human thighs after one-legged immobilization. Stuart CA, Shangraw RE, Prince MJ, et al. Bed-rest-induced insulin resistance occurs primarily in muscle. Effect of oral creatine supplementation on human muscle GLUT4 protein content after immobilization. Bergouignan A, Momken I, Schoeller DA, et al.

Regulation of energy balance during long-term physical inactivity induced by bed rest with and without exercise training. McBeath AA, Bahrke M, Balke B. Efficiency of assisted ambulation determined by oxygen consumption measurement.

Waters RL, Campbell J, Perry J. Energy cost of three-point crutch ambulation in fracture patients. J Orthop Trauma. Frankenfield D. Energy expenditure and protein requirements after traumatic injury. Biolo G, Ciocchi B, Stulle M, et al.

Calorie restriction accelerates the catabolism of lean body mass during 2 wk of bed rest. Mettler S, Mitchell N, Tipton KD. Increased protein intake reduces lean body mass loss during weight loss in athletes.

Med Sci Sports Exerc. Wolfe RR. The underappreciated role of muscle in health and disease. Pasiakos SM, Vislocky LM, Carbone JW, et al.

Acute energy deprivation affects skeletal muscle protein synthesis and associated intracellular signaling proteins in physically active adults. J Nutr. Areta JL, Burke LM, Camera DM, et al. Reduced resting skeletal muscle protein synthesis is rescued by resistance exercise and protein ingestion following short-term energy deficit.

Forbes GB, Brown MR, Welle SL, et al. Deliberate overfeeding in women and men: energy cost and composition of the weight gain. Br J Nutr. Biolo G, Agostini F, Simunic B, et al. Positive energy balance is associated with accelerated muscle atrophy and increased erythrocyte glutathione turnover during 5 wk of bed rest.

Walhin JP, Richardson JD, Betts JA, et al. Exercise counteracts the effects of short-term overfeeding and reduced physical activity independent of energy imbalance in healthy young men.

Stephens FB, Chee C, Wall BT, et al. Lipid induced insulin resistance is associated with an impaired skeletal muscle protein synthetic response to amino acid ingestion in healthy young men.

Rebello CJ, Liu AG, Greenway FL, et al. Dietary strategies to increase satiety. Adv Food Nutr Res. Arnold M, Barbul A. Nutrition and wound healing. Plast Reconstr Surg. Demling RH. Nutrition, anabolism, and the wound healing process: an overview.

PubMed Central PubMed Google Scholar. Quevedo MR, Price GM, Halliday D, et al. Nitrogen homoeostasis in man: diurnal changes in nitrogen excretion, leucine oxidation and whole body leucine kinetics during a reduction from a high to a moderate protein intake. Trappe TA, Burd NA, Louis ES, et al.

Influence of concurrent exercise or nutrition countermeasures on thigh and calf muscle size and function during 60 days of bed rest in women. Biolo G, Tipton KD, Klein S, et al. An abundant supply of amino acids enhances the metabolic effect of exercise on muscle protein.

Witard OC, Jackman SR, Breen L, et al. Myofibrillar muscle protein synthesis rates subsequent to a meal in response to increasing doses of whey protein at rest and after resistance exercise.

Moore DR, Robinson MJ, Fry JL, et al. Ingested protein dose response of muscle and albumin protein synthesis after resistance exercise in young men.

Article PubMed CAS Google Scholar. Yang Y, Breen L, Burd NA, et al. Resistance exercise enhances myofibrillar protein synthesis with graded intakes of whey protein in older men. Areta JL, Burke LM, Ross ML, et al.

Timing and distribution of protein ingestion during prolonged recovery from resistance exercise alters myofibrillar protein synthesis.

Mamerow MM, Mettler JA, English KL, et al. Dietary protein distribution positively influences h muscle protein synthesis in healthy adults. Burke LM, Slater G, Broad EM, et al.

Eating patterns and meal frequency of elite Australian athletes. Int J Sport Nutr Exerc Metab. PubMed Google Scholar. Garcia-Roves PM, Fernandez S, Rodriguez M, et al. Eating pattern and nutritional status of international elite flatwater paddlers.

Tipton KD, Ferrando AA, Phillips SM, et al. Postexercise net protein synthesis in human muscle from orally administered amino acids. Tipton KD, Gurkin BE, Matin S, et al. Nonessential amino acids are not necessary to stimulate net muscle protein synthesis in healthy volunteers.

J Nutr Biochem. Paddon-Jones D, Sheffield-Moore M, Urban RJ, et al. Essential amino acid and carbohydrate supplementation ameliorates muscle protein loss in humans during 28 days bedrest.

Bostock EL, Pheasey CM, Morse CI, et al. Effects of essential amino acid supplementation on muscular adaptations to 3 weeks of combined unilateral glenohumeral and radiohumeral joints immobilisation.

J Athl Enhanc. doi: Brooks N, Cloutier GJ, Cadena SM, et al. Resistance training and timed essential amino acids protect against the loss of muscle mass and strength during 28 days of bed rest and energy deficit.

Dreyer HC, Strycker LA, Senesac HA, et al. Essential amino acid supplementation in patients following total knee arthroplasty. J Clin Invest. Kimball SR. Regulation of global and specific mRNA translation by amino acids. Kimball SR, Jefferson LS. Role of amino acids in the translational control of protein synthesis in mammals.

Semin Cell Dev Biol. Wilkinson DJ, Hossain T, Hill DS, et al. Effects of leucine and its metabolite beta-hydroxy-beta-methylbutyrate on human skeletal muscle protein metabolism. Nicastro H, Artioli GG, Costa Ados S, et al.

An overview of the therapeutic effects of leucine supplementation on skeletal muscle under atrophic conditions. Amino Acids. Anthony JC, Anthony TG, Layman DK.

Leucine supplementation enhances skeletal muscle recovery in rats following exercise. Lang CH, Frost RA, Deshpande N, et al. Alcohol impairs leucine-mediated phosphorylation of 4E-BP1, S6K1, eIF4G, and mTOR in skeletal muscle.

Baptista IL, Leal ML, Artioli GG, et al. Leucine attenuates skeletal muscle wasting via inhibition of ubiquitin ligases. Muscle Nerve. Stein TP, Donaldson MR, Leskiw MJ, et al.

Branched-chain amino acid supplementation during bed rest: effect on recovery. Katsanos CS, Kobayashi H, Sheffield-Moore M, et al.

A high proportion of leucine is required for optimal stimulation of the rate of muscle protein synthesis by essential amino acids in the elderly. Rieu I. Leucine supplementation improves muscle protein synthesis in elderly men independently of hyperaminoacidaemia.

Wall BT, Hamer HM, de Lange A, et al. Leucine co-ingestion improves post-prandial muscle protein accretion in elderly men. Clin Nutr. Hespel P, Derave W. Ergogenic effects of creatine in sports and rehabilitation. Subcell Biochem. Tarnopolsky MA. Clinical use of creatine in neuromuscular and neurometabolic disorders.

Oral creatine supplementation facilitates the rehabilitation of disuse atrophy and alters the expression of muscle myogenic factors in humans.

Roy BD, de Beer J, Harvey D, et al. Creatine monohydrate supplementation does not improve functional recovery after total knee arthroplasty. Arch Phys Med Rehabil. Johnston AP, Burke DG, MacNeil LG, et al.

Effect of creatine supplementation during cast-induced immobilization on the preservation of muscle mass, strength, and endurance. J Strength Cond Res. Calder PC, Albers R, Antoine JM, et al. Inflammatory disease processes and interactions with nutrition.

Calder PC. n-3 Fatty acids, inflammation and immunity: new mechanisms to explain old actions. Albina JE, Gladden P, Walsh WR. Detrimental effects of an omega-3 fatty acid-enriched diet on wound healing. J Parenter Enteral Nutr.

Otranto M, Do Nascimento AP, Monte-Alto-Costa A. Effects of supplementation with different edible oils on cutaneous wound healing. Wound Repair Regen. You J-S, Park M-N, Song W, et al.

Dietary fish oil alleviates soleus atrophy during immobilization in association with Akt signaling to p70s6k and E3 ubiquitin ligases in rats.

Smith GI, Atherton P, Reeds DN, et al. Dietary omega-3 fatty acid supplementation increases the rate of muscle protein synthesis in older adults: a randomized controlled trial.

Omega-3 polyunsaturated fatty acids augment the muscle protein anabolic response to hyperinsulinaemia-hyperaminoacidaemia in healthy young and middle-aged men and women. McGlory C, Galloway SD, Hamilton DL, et al.

Temporal changes in human skeletal muscle and blood lipid composition with fish oil supplementation. Prostaglandins Leukot Essent Fatty Acids. You J-S, Park M-N, Lee Y-S. Dietary fish oil inhibits the early stage of recovery of atrophied soleus muscle in rats via Akt—p70s6k signaling and PGF2α.

Barker T, Martins TB, Hill HR, et al. Low vitamin D impairs strength recovery after anterior cruciate ligament surgery. J Evid Based Complement Altern Med. Magne H, Savary-Auzeloux I, Remond D, et al. Nutritional strategies to counteract muscle atrophy caused by disuse and to improve recovery.

Nutr Res Rev. Barker T, Leonard SW, Hansen J, et al. Vitamin E and C supplementation does not ameliorate muscle dysfunction after anterior cruciate ligament surgery. Free Radic Biol Med. Amen DG, Newberg A, Thatcher R, et al. Impact of playing American professional football on long-term brain function.

J Neuropsychiatry Clin Neurosci. Guskiewicz KM, Marshall SW, Bailes J, et al. Association between recurrent concussion and late-life cognitive impairment in retired professional football players. Barrett EC, McBurney MI, Ciappio ED.

Adv Nutr. Wu A, Ying Z, Gomez-Pinilla F. Dietary curcumin counteracts the outcome of traumatic brain injury on oxidative stress, synaptic plasticity, and cognition.

Exp Neurol. Mills JD, Hadley K, Bailes JE. Dietary supplementation with the omega-3 fatty acid docosahexaenoic acid in traumatic brain injury. Wang T, Van KC, Gavitt BJ, et al. Effect of fish oil supplementation in a rat model of multiple mild traumatic brain injuries.

Restor Neurol Neurosci. The salutary effects of DHA dietary supplementation on cognition, neuroplasticity, and membrane homeostasis after brain trauma. J Neurotrauma. Exercise facilitates the action of dietary DHA on functional recovery after brain trauma.

Dietary strategy to repair plasma membrane after brain trauma: implications for plasticity and cognition. Neurorehabil Neural Repair. Lewis M, Ghassemi P, Hibbeln J. Therapeutic use of omega-3 fatty acids in severe head trauma. Am J Emerg Med.

Video

What I Eat in a Day No Planning, Easy Plant Based Meals Click name to view affiliation. Injuries are an injurry consequence of athletic performance with preventiln Turmeric sustaining one or more during their DKA complications careers. As many Nutritional support for injury prevention Enhancing concentration in sports in preventioh athletes Alpha-lipoic acid and blood sugar control an injury during international competitions, Nutritiojal of which result in time lost from training and competition. Other common injuries include fractures, especially stress fractures in athletes with low energy availability, and injuries to tendons and ligaments, especially those involved in high-impact sports, such as jumping. Given the high prevalence of injury, it is not surprising that there has been a great deal of interest in factors that may reduce the risk of injury, or decrease the recovery time if an injury should occur: One of the main variables explored is nutrition. Nutritional support for injury prevention

Author: Yozshuk

3 thoughts on “Nutritional support for injury prevention

  1. Ich entschuldige mich, aber meiner Meinung nach sind Sie nicht recht. Geben Sie wir werden besprechen. Schreiben Sie mir in PM, wir werden reden.

Leave a comment

Yours email will be published. Important fields a marked *

Design by ThemesDNA.com