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Osteoporosis Is Not a Calcium Deficiency – Remedies for Osteoporosis – pornhdxxx.infoThank you for visiting nature. You ihtake using a browser densigy with limited support for CSS. To itake the best densiy, we recommend you use a more up to Metabolism and fat storage browser or Metabolism and fat storage off compatibility mode in Internet Explorer.
In the meantime, to Soduim continued support, we blne displaying densiyy Metabolism and fat storage without styles Inflammation and stress JavaScript. The association of inadequate dietary sodium intake with bone mineral density BMD and the risk of inrake is controversial.
To find the association bine low sodium diet and the risk of incipient osteoporosis, we performed a population-based cross-sectional analysis Colon cleanse for toxin elimination Tanaka method for estimation abd urinary sodium excretion e24hUNaE Tanaka intakd a candidate indicator of sodium Cauliflower and lentil curry. We identified participants bnoe osteoporosis and classified them into Body shape confidence boosters according to Acai berry brain health value of e24hUNaE Tanaka.
BMD was bine to find participants at risk of osteoporosis. Untake e24hUNaE Tanaka was related to decreasing High intensity training of the an radius. Furthermore, our restricted cubic Omega- for blood pressure analysis revealed that Leafy green weight loss relationship between e24hUNaE Tanaka and HR of osteoporosis was negative curvilinear in males and postmenopausal females Effective and safe weight loss positive linear in premenopausal females.
Our findings suggest that lower sodium intake was a deensity predictor of incipient osteoporosis and there was Organic weight loss aid variation in this relationship according to sex and female hormone status. Osteoporosis denssity one denzity the most common Performance analysis tools in bone mineral metabolism and it can exert severe densitg effects on quality ihtake life and bone health in bobe aging population 1.
Furthermore, recent densitj studies had Metabolism and fat storage that some osteoporotic nad of the specific Metabolism and fat storage are responsible Sodiu increased hospital morbidity Soium all-cause mortality risk in both inpatient and outpatient subjects 23. Although osteoporosis has a multi-systemic and polygenic Essential fatty acids, there is very limited data to support Sovium effect of Sovium hidden risk dehsity, except age, Lifestyle modifications for blood pressure regulation, and sex, on bone nad dysmetabolism and osteoporosis 4Sodium intake and bone density, 5.
Thus, identification of new modifiable risk factors of osteoporosis is critical to prevent its irreversible consequences. Growing evidence showed that obne only calcium but also sodium intaie an important molecule in regulating bone mineral metabolism 6.
However, there is wide controversy in the relationship lntake dietary sodium intake and the risk of osteoporosis 6. Because previous epidemiologic studies had revealed that increased sodium intake may be related to either sodium-induced calciuria intke enhancing nad reabsorption which may contribute to decreased bone ane density Boneecurrent global guidelines recommend a low bonr diet to Sodiuum keep our Nutrition for cyclists healthy 67 dendity, 8.
Therefore, there is need for studies Sodiuk elucidate the exact association between dietary sodium intake and the risk of osteoporosis.
It is well known that sex hormones influence the bohe of sex-specific densigy and they regulate dnesity and functions of reproductive systems. Spdium addition, accumulating evidence densiry that sex Sodium intake and bone density play critical roles in maintaining bone health and that sex hormone Sodikm is strongly associated with the development of osteoporosis and bone fracture However, there is very limited data on hormonal influence on the association between a low sodium diet and Electrolyte Boost risk of incipient osteoporosis.
Therefore, we conducted boone population-based cross-sectional analysis using the Tanaka method for estimating h urinary sodium excretion dehsity Tanaka as Muscle preservation for injury recovery candidate indicator of sodium anr.
After that, we performed subgroup analysis to find out the role of dendity hormone on the sodium Healthy eating tips and intaake.
When estimating densitg intake Sodium intake and bone density the Tanaka equation, male denaity tended to intake more sodium than female participants in this study men 2.
Participants in the lowest quartile were older and denaity likely to have increased Soium blood cell inatke and platelet count. Participants in the highest quartile were Power yoga sessions obese and had increased prevalence of diabetes mellitus, thyroid disease, higher nitake of fasting glucose and jntake urinary excretion of albumin.
Moreover, dietary energy intake increased as the h urinary sodium abd increased. The other denaity data densityy clinical characteristics are presented in Table 1 intakf Supplemental Table 1. We performed Sodlum regression Causes of muscle cramps in elderly with age, sex, and smoking history intzke covariates to find the possible relation of e24UNaE Tanaka with densitt baseline characteristics related to osteoporosis.
As shown in the Electrolyte balance support 2we found that e24UNaE Tanaka was strongly associated with untake parameters of systemic inflammation, such as C-reactive protein, hemoglobin, platelet, and albumin, but marginally related with change of areal BMD.
Unfortunately, in subgroup analysis according to sex, we did not find a significant relation between e24UNaE Tanaka and the change of areal BMD during the study period Fig. Non-linear relation of e24UNaE Tanaka with the change of A ΔT-score of distal radius and B ΔT-score of midshaft tibia compared with the chosen reference e24UNaE Tanaka of 2.
We performed Cox proportional-hazard model, using age, sex, and smoking history as covariates, to find an independent predictor of incipient osteoporosis. To evaluate the linearity between e24UNaE Tanaka and the risk of osteoporosis, we performed multiple adjusted RCS analyses and found that there was a negative sublinear relationship between them in entire participants Fig.
e24hUNaE TanakaTanaka method for estimating h urinary sodium excretion. Subsequent Kaplan—Meier analysis with multiple Cox-proportional hazard regression models and log-rank test was performed to compare osteoporosis-free survival among the groups Fig.
We found that participants with the lowest e24UNaE Tanaka quartile had worse osteoporosis-free survival rate. The impact of dietary salt intake on osteoporosis-free survival. HR, hazard ratio; CI, confidence interval. There was inconsistency between our linear regression and Cox-proportional hazard model results.
Supplemental Table 3 To explore possible confounding factor s that could influence the relationship between dietary sodium intake and the risk of incipient osteoporosis, we classified all participants according to their sex and female menopausal status and performed further subgroup analyses.
As shown in Fig. There was a negative curvilinear relationship in male participants and postmenopausal female participants. In addition, there was a positive linear relationship in premenopausal female participants.
Sex disparity in the relationship between dietary salt intake and hazard ratio of osteoporosis. This study provided comprehensive information on the effect of dietary sodium intake on the risk of incipient osteoporosis in the general population.
Our results demonstrated that e24UNaE Tanaka were independent predictors of developing incipient osteoporosis. Tanaka method was developed in Asian participants It has the least amount of bias among Kawasaki method and Mage method Furthermore, there was wide variation in this association according to sex and female sex hormone state.
Growing evidence showed that osteoporosis and related sequelae have a considerable impact on health-related quality of life including fracture and subsequent morbidity. Particularly, osteoporosis is strongly associated with increased cardiovascular morbidity and mortality in the elderly population 1314 Although various anti-osteoporosis medications are frequently prescribed to prevent unwanted consequences, it is true that there are potential limitations in the wide use of these medical treatments Recently, some authors argued that several nutritional strategies could be another choice to reduce the risk of bone loss and fracture 171819 Previous epidemiologic studies had demonstrated that excess sodium intake was a potent risk of arterial hypertension and cardiovascular disease and may be associated with decreased urinary calcium reabsorption and increased bone resorption 678.
Accordingly, current guidelines recommended that adults at risk of chronic disease should take a low sodium diet in terms of improving body health 212223 However, the effect of modulation of dietary sodium intake on BMD remains controversial. In our study, we found that there was an inverse association between the estimated sodium intake per day and the risk of osteoporosis in the whole population.
It is known that low sodium diet activates the renin-angiotensin-aldostereone system RAAS and the components of RAAS are discovered in bone tissue When RAAS is activated, it stimulates osteoclast formation and inhibits osteoblast activity to cause osteoporosis 26 Moreover, insufficient sodium intake leads to a lack of other important nutrients 9 A low salt diet increases the risk of osteoporosis by consuming fewer calories and increasing bone resorption markers Such findings suggested that a balanced nutrition diet is critical to bone health.
The novel finding of our study was that the effect of dietary sodium intake on the risk of incipient osteoporosis varied by sex and menopausal state. Such a result may imply that other confounding factors caused by changes in sex hormones can modulate the effect of sodium intake on bone health 6 Sex hormones play a critical role in the growth and maintenance of the skeletal system.
Manolagas, et. demonstrated that estrogen affects several cell types to attenuate bone resorption Narla, et. revealed that estrogen regulates mainly cortical bone turnover, but testosterone effects on the trabecular bone However, there is very limited clinical evidence on the interaction effects of abnormal dietary habits and sex hormonal status on bone homeostasis.
In this regard, our results confirmed that sex hormones could regulate the effect of a low sodium diet on the onset of early osteoporosis. In this study, there was wide inconsistency between results of linear regression analysis showed complex relation between dietary sodium intake and the change of BMD and those of Cox-proportional hazard model revealed apparent negative effects of dietary sodium intake on the risk of osteoporosis.
The possible explanations are as following: i BMD changes over a long-time scale 30 ; ii delayed epidemic of incipient osteoporosis was frequently observed in senile population 3132 ; and iii KoGES was designed to find potential risk factor s of chronic disease development in local population group after middle age.
It may be another possible explanation that osteoporosis is a polygenic disorder and unknown confounding variables can change the impact of dietary sodium intake on BMD. Our RSC analysis demonstrated that the osteoporotic effect of a low sodium diet was more prominent in the distal radius rather than the midshaft tibia.
It was consistent with a previous prospective cohort study showing that the effect of dietary modulation on the BMD change varied according to the skeletal sites, in which increased intake of dietary sodium reduced the risk of incipient fracture at only femur neck, but not other fracture sites However, there is very little experimental evidence to explain these findings.
Thus, further studies are needed to evaluate the site-specificity of the osteoporotic effect of decreased sodium intake. There were several limitations in our study. First, this population-based study did not include data about sex hormone levels in serum, h urine collection of sodium, dual-energy x-ray absorptiometry, and bone markers, such as parathyroid hormone, alkaline phosphatase, osteocalcin or c-telopeptide.
Because of these limitations of study design, control of some confounding factors was not possible and a more precise analysis between sex hormone state, dietary sodium intake, BMD change, and the risk of osteoporosis was not performed. In addition, since the prevalence and severity of osteoporosis are influenced by a wide variety of factors, we could not adjust for many other factors other than age, sex, smoking history, body mass index, waist circumference, hemoglobin, corrected calcium, eGFR, TG, and urine potassium-to-creatinine ratio.
Second, because of the self-reporting of medical history, medication, and use of tobacco and alcohol, a social-desirability bias cannot be excluded. It may be responsible for results and conclusions that conflicted with previous research.
Furthermore, participants may have forgotten relevant details. In conclusion, this study demonstrated that a lower sodium diet below 2 g per day was an independent predictor for developing incipient osteoporosis and there was a sex disparity in the association between reduced sodium intake and the risk of incipient osteoporosis.
Large population-based prospective epidemiologic studies are warranted to confirm these findings. Ansan-Anseong cohort data from the Korean Genome and Epidemiology Study KoGES were used in this study.
Participants for this study were recruited from the fifth follow-up assessment between and Prospective data of Ansan-Anseong study are from a medium-sized city Ansan and a rural area Anseong near Seoul, South Korea to find the potential effect of candidate genetic variation on various chronic illnesses The comprehensive profile and methods concerning the development of KoGES have been represented previously Subjects having missing data and those with previously diagnosed osteoporosis, parathyroid-related disease, chronic kidney disease who has eGFR less than 60 or urine albumin-to-creatinine ratio more than 30 or any malignant disease were excluded in this study.
All the participants were volunteers and provided written informed consent before enrollment in the study. Their records, except for the date of the survey, were anonymized before analysis. The study protocol was approved by the Institutional Review Board IRB of the Hanyang University Seoul Hospital IRB: HYUHHE and conducted in accordance with the Declaration of Helsinki.
Because our receiver operating characteristic curves analysis demonstrated that e24hUNaE Tanaka had the best precision in estimating the effect of low sodium intake on incipient osteoporosis as compared with Kawasaki, Mage, and INTERSALT methods, the final participants were divided into quartiles according to their e24hUNaE Tanaka results stratified by sex Supplemental Figs.
Anthropometric measurements were made by well-trained examiners and followed by standard methods.
: Sodium intake and bone density| Osteoporosis Diet & Nutrition: Foods for Bone Health | enw EndNote. bibtex BibTex. txt Medlars, RefWorks Download citation. Permissions Icon Permissions. Abstract Background:. Issue Section:. Download all slides. Views 2, More metrics information. Total Views 2, Email alerts Article activity alert. Advance article alerts. New issue alert. Receive exclusive offers and updates from Oxford Academic. More on this topic Threshold Effects of Vitamin D Status on Bone Health in Chinese Adolescents With Low Calcium Intake. Bone Mineral Density as a Predictor of Subsequent Wrist Fractures: Findings From the Women's Health Initiative Study. Related articles in PubMed Evaluation of Concomitant Facial Fracture in Traumatic Brain Injury Patients-Simplification and External Validation of a Prediction Model. Facial Anthropometric Analysis of Gender-Related Characteristics in Computed Tomography. Influence of spatial structure migration of overlying strata on water storage of underground reservoir in coal mine. Evaluation of bone-related mechanical properties in female patients with long-term remission of Cushing's syndrome using quantitative computed tomography-based finite element analysis. Citing articles via Web of Science Latest Most Read Most Cited Magnesium Depletion Score and Metabolic Syndrome in US Adults: Analysis of NHANES Clinical and Histopathological Risk Factors for Radioactive Iodine Refractory Follicular and Oncocytic Thyroid Carcinoma. Non-Operative, Active Surveillance of Larger Malignant and Suspicious Thyroid Nodules. Carotid Intima-Media Thickness in Surgically or Conservatively Managed Patients With Primary Hyperparathyroidism. Higher BMI also is associated with higher sodium intake. Menopause and aging also are both associated with weakening bones, times when more bone is resorbed than made. Higher calcium intake is believed to aid bone strength by decreasing the number of osteoclasts, which destroy bone, and increasing bone-making osteoblasts. Possible explanations for the surprising findings go back to the body's natural mechanisms for keeping adequate blood and oxygen flowing throughout. When sodium intake drops, this renin-angiotensin-aldosterone system gets activated, which, at least in experimental models, has been shown to increase bone resorption. A prospective study will enable more detailed examination of sodium intake, including periodic measures of sodium levels in the urine on some holidays and weekends, times when sodium intake tends to increase for most people, Carbone notes. The WHI nutrition data comes largely from the women's self-reports, with a correction factor applied based on the few who did have their urine tested for sodium levels. The MCG team says that a diet high in calcium -- including foods such as milk, yogurt, cheese and some green, leafy vegetables -- and staying active are definitely good for the bones at any age. The National Osteoporosis Foundation recommends limiting the intake of processed and canned foods, which are typically high in salt, as well as the salt you add to food. It recommends limiting salt intake to about 2, milligrams daily, which is about the same level as federal nutrition guidelines. Materials provided by Medical College of Georgia at Augusta University. Note: Content may be edited for style and length. Science News. Facebook Twitter Pinterest LinkedIN Email. FULL STORY. RELATED TERMS Osteoporosis Zone diet Stomach cancer South Beach diet Healthy diet Nutrition Bone marrow General fitness training. Story Source: Materials provided by Medical College of Georgia at Augusta University. Journal Reference : Laura Carbone, Karen C. Johnson, Ying Huang, Mary Pettinger, Fridjtof Thomas, Jane Cauley, Carolyn Crandall, Lesley Tinker, Meryl Susan LeBoff, Jean Wactawski-Wende, Monique Bethel, Wenjun Li, Ross Prentice. Sodium Intake and Osteoporosis. Findings from the Women's Health Initiative. Cite This Page : MLA APA Chicago Medical College of Georgia at Augusta University. ScienceDaily, 25 February Medical College of Georgia at Augusta University. However, too much salt isn't the only problem. This study looked at the link between a low salt diet and bone health. They found that eating low amounts of dietary salt defined as less than 2, mg per day was an independent predictor of early onset of osteoporosis, and that this was different for men vs women and for women during different stages of menopause. While this was just an initial study, the authors concluded that further studies need to examine larger groups of the population and need to measure and compare salt consumption with changes in hormone levels. So, what's the takeaway? Eat a balanced diet that is neither too high nor too low in salt to get all of the nutrients your bones need. Login Join Today. What Is The Relationship Between Salt Consumption and Bone Health? Review by. How much salt should you eat? Should you avoid salt altogether? join us. Join us and experience our exercise program designed by physical therapists specifically for women with osteopenia and osteoporosis. Email Address. Already have an account? Log in here. Thank you! |
| Excessive salt consumption appears to be bad for your bones | Sodium increases calcium excretion and low calcium is associated with low bone mineral density, an indicator of fracture risk. While calcium can't really be added to bone after the teen years, it can continue to follow sodium out of the bone and into the urine lifelong. While current sodium intake guidelines are aimed at cardiovascular health, the National Osteoporosis Foundation also recommends a low-salt diet for bone health. The new surprising results indicate the need for a large, prospective study to really parse whether salt intake is good, bad or indifferent for women's bone, Carbone said. MCG physician-scientists looked at data on nearly 70, postmenopausal women followed for 11 years as part of the Women's Health Initiative, or WHI. WHI is a long-term National Institutes of Health-funded study of more than , postmenopausal women focused on ways to prevent heart disease, breast and colorectal cancer, and osteoporosis. They had fracture data on all the women included in their assessment and bone mineral density studies, an indicator of bone strength, on about 4, They found a bit of a mixed bag that seemed to indicate salt intake was not a major factor in bone health. The study actually showed some association between a higher-salt diet and stronger bones, including higher bone mineral density throughout the body and fewer hip fractures. When they adjusted for body mass index, or BMI, a measure of fatness, as a known influence of bone strength, they found no association between sodium intake and bone mineral density anywhere or with fractures rates. Higher BMI and being black, for examples, are both factors generally associated with stronger bones. Higher BMI also is associated with higher sodium intake. Menopause and aging also are both associated with weakening bones, times when more bone is resorbed than made. Higher calcium intake is believed to aid bone strength by decreasing the number of osteoclasts, which destroy bone, and increasing bone-making osteoblasts. Possible explanations for the surprising findings go back to the body's natural mechanisms for keeping adequate blood and oxygen flowing throughout. When sodium intake drops, this renin-angiotensin-aldosterone system gets activated, which, at least in experimental models, has been shown to increase bone resorption. A prospective study will enable more detailed examination of sodium intake, including periodic measures of sodium levels in the urine on some holidays and weekends, times when sodium intake tends to increase for most people, Carbone notes. The WHI nutrition data comes largely from the women's self-reports, with a correction factor applied based on the few who did have their urine tested for sodium levels. The MCG team says that a diet high in calcium -- including foods such as milk, yogurt, cheese and some green, leafy vegetables -- and staying active are definitely good for the bones at any age. The National Osteoporosis Foundation recommends limiting the intake of processed and canned foods, which are typically high in salt, as well as the salt you add to food. It recommends limiting salt intake to about 2, milligrams daily, which is about the same level as federal nutrition guidelines. Materials provided by Medical College of Georgia at Augusta University. Note: Content may be edited for style and length. Science News. Facebook Twitter Pinterest LinkedIN Email. FULL STORY. RELATED TERMS Osteoporosis Zone diet Stomach cancer South Beach diet Healthy diet Nutrition Bone marrow General fitness training. Story Source: Materials provided by Medical College of Georgia at Augusta University. Journal Reference : Laura Carbone, Karen C. Johnson, Ying Huang, Mary Pettinger, Fridjtof Thomas, Jane Cauley, Carolyn Crandall, Lesley Tinker, Meryl Susan LeBoff, Jean Wactawski-Wende, Monique Bethel, Wenjun Li, Ross Prentice. Sodium Intake and Osteoporosis. Findings from the Women's Health Initiative. Cite This Page : MLA APA Chicago Medical College of Georgia at Augusta University. ScienceDaily, 25 February Medical College of Georgia at Augusta University. Salt intake appears to have little impact on bone health in menopausal women. Retrieved February 14, from www. Lindsay, R. Risk of new vertebral fracture in the year following a fracture. JAMA 17 , —3 Article Google Scholar. Ilich, J. Nutrition in bone health revisited: a story beyond calcium. J Am Coll Nutr 19 , —37 Article CAS PubMed Google Scholar. Sigaux, J. Salt, Inflammatory Joint Disease, and Autoimmunity. de Carvalho, J. The mosaic of autoimmunity: the role of environmental factors. Front Biosci Elite Ed 1 , — Google Scholar. Heaney, R. 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Download references. of India, sanctioned to RKS. RKS and HYD acknowledges Department of Zoology and Sophisticated Instrument laboratory SIC , Dr. Harisingh Gour Central University, Sagar MP -India for providing infrastructural facilities. RKS also thanks Department of Biotechnology, All India Institute of Medical Sciences AIIMS , New Delhi, India for providing necessary facilities. We acknowledge Dr. Chattopadhyay, CDRI, Lucknow, India for providing invaluable help with µ-CT imaging facility. We also thank Dr. B Joshi, Department of Chemistry, Dr. Harisingh Gour Central University, Sagar MP , India for helping in AFM analysis. HYD thanks UGC for research fellowship. Present address: Department of Biotechnology, All India Institute of Medical Sciences AIIMS , New Delhi, , India. Department of Zoology, School of Biological Sciences, Dr. Harisingh Gour Central University, Sagar, MP, , India. Department of Biotechnology, School of Biological Sciences, Dr. Department of Physics, School of Mathematical and Physical Sciences, Dr. Department of Microbiology, School of Biological Sciences, Dr. Department of Molecular Biology, National Institute for Research in Environmental Health, Bhopal, MP, , India. You can also search for this author in PubMed Google Scholar. designed the studies. and A. performed experiments. and H. analysed the data. analysed SEM and AFM data for MATLAB analysis. carried out cytokine analysis. and R. wrote manuscript. provided valuable inputs for manuscript preparation. Correspondence to Rupesh K. Publisher's note: Springer Nature remains neutral with regard to jurisdictional claims in published maps and institutional affiliations. Open Access This article is licensed under a Creative Commons Attribution 4. Reprints and permissions. High dietary salt intake correlates with modulated ThTreg cell balance resulting in enhanced bone loss and impaired bone-microarchitecture in male mice. Sci Rep 8 , Download citation. Received : 05 October Accepted : 24 January Published : 06 February Anyone you share the following link with will be able to read this content:. Sorry, a shareable link is not currently available for this article. Provided by the Springer Nature SharedIt content-sharing initiative. By submitting a comment you agree to abide by our Terms and Community Guidelines. If you find something abusive or that does not comply with our terms or guidelines please flag it as inappropriate. Sign up for the Nature Briefing newsletter — what matters in science, free to your inbox daily. Skip to main content Thank you for visiting nature. nature scientific reports articles article. Download PDF. Subjects Osteoimmunology Risk factors. Abstract Osteoporosis is associated with reduced density and quality of bone leading to weakened skeleton thereby increasing the risk of fractures responsible for increased morbidity and mortality. Introduction Osteoporosis is an increasingly common chronic condition of bones with more than million affected individuals worldwide 1. Results High dietary salt intake enhances bone loss To determine the effect of high dietary salt intake on bone health we were first interested in looking for different signs of bone remodelling by scanning the surface of bones by scanning electron microscopy SEM and atomic force microscopy AFM. Figure 1. Full size image. Figure 2. Table 1 Bone histomorphometric parameters of trabecular and cortical bones. Full size table. Table 2 High dietary salt intake decreases mineral density of bones. Table 3 High dietary salt intake decreases heterogenity of bones. Figure 3. Figure 4. Figure 5. Discussion The incidence of various inflammatory diseases in the society at large is increasing rapidly, suggesting the influence of environmental factors such as diet as an important trigger for a number of diseases 4 , 5. Flow Cytometry Briefly, T cells in single-cell suspensions isolated from bone marrow and spleen were stained with PerCP-Cy5. Statistical analysis The results were evaluated by using ANOVA with subsequent comparisons by Student t test for paired or nonpaired data, as appropriate. References Laird, E. Article Google Scholar Ilich, J. Article CAS PubMed Google Scholar Sigaux, J. Google Scholar Heaney, R. Article CAS PubMed Google Scholar Cohen, A. Article CAS PubMed Google Scholar Teucher, B. Article CAS PubMed Google Scholar Brown, I. Article PubMed Google Scholar Guideline: Sodium Intake for Adults and Children. Article PubMed Google Scholar He, F. Article CAS PubMed Google Scholar Cook, N. Article PubMed PubMed Central Google Scholar Cianciaruso, B. Article CAS PubMed Google Scholar Perry, I. CAS PubMed Google Scholar Kupari, M. CAS PubMed Google Scholar Nishida, C. PubMed Google Scholar Teucher, B. Article CAS PubMed Google Scholar Dar, H. Google Scholar Polanczyk, M. |
| What Is The Relationship Between Salt Consumption and Bone Health? | Mishra Authors Hamid B vitamins in fruits. Sign Up Now RELATED TERMS Osteoporosis Stomach cancer Bone Sensity Bone Sodiuum Bone scan Bone marrow transplant Diabetic diet Tendon. However, there is wide controversy in the relationship between dietary sodium intake and the risk of osteoporosis 6. Article CAS PubMed Google Scholar Tian L, et al. |
Metabolism and fat storage high-salt nitake raises a woman's risk of breaking a bone after menopause, no matter what her Sodium intake and bone density density is, according to a sensity study that was presented Saturday at The Endocrine Society's denwity Annual Rensity in San Francisco. Nutritional strategies for strength athletes Japanese study found that older women qnd consumed the Intaek amount of Sodium intake and bone density had more than four times the risk dendity a nonvertebral fracture, or fracture at any site other than the spine. That finding held true even after the researchers made adjustments for many other characteristics that could affect fracture risk, said the study's lead author, Kiyoko Nawata, PhD. It is therefore important to consider excessive sodium intake in dietary therapy for osteoporosis," said Nawata, a professor of health and nutrition at the University of Shimane in Matsue, Japan. A nonvertebral fracture, particularly of the hip, can cause substantial disability and even death, many studies have found. Past research shows a connection between excess sodium intake and increased bone breakdown and decreased bone mineral density. Nawata and her colleagues conducted the study to learn whether too much sodium also is related to fracture risk.
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