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Your clma may be very Healthy caloric intake to touch—even a Probiotics for urinary tract health sheet can hurt. These are all symptoms Diwbetic peripheral nerve damage.
Damafe generally Healthy caloric intake in the feet, usually in both feet at nwrve. Sometimes amputation removal by surgery is necessary. Finding and treating foot problems early can lower your chances of developing a serious infection. Learn how to care for your feetincluding how to check them yourself and what kind of shoes to wear.
Nerve damage can cause muscles in your digestive tract to slow down or stop working. Learn more about how diabetes can affect your digestion. Autonomic nerve damage affects your heart, bladder, stomach, intestines, sex organs, or eyes. Symptoms may include:.
Proximal nerve damage affects nerves in the thighs, hips, buttocks, or legs. It can also affect the stomach and chest area. Focal nerve damage affects single nerves, most often in your hand, head, torso, or leg.
Keeping your blood sugar as close to your target range as possible is the best way to help prevent or delay nerve damage. Other things you can do are:. Most people with diabetes can prevent serious nerve damage complications.
Stay on schedule with all of your self-checks, exams, and appointments with a diabetes care schedule. Skip directly to site content Skip directly to page options Skip directly to A-Z link. Español Other Languages. Diabetes and Nerve Damage. Español Spanish. Minus Related Pages.
Nerve damage can affect your hands, feet, legs, and arms. Nerve Damage and Digestion. Risk Factors for Nerve Damage. Anyone with diabetes can develop nerve damage, but these factors increase your risk: Blood sugar levels that are hard to manage.
Having diabetes for a long time, especially if your blood sugar is often higher than your target levels. Being overweight. Being older than Having high blood pressure.
Having high cholesterol. Learn More. Diabetes and Digestion Diabetes Care Schedule Diabetes and Your Feet CDC Diabetes on Facebook CDCDiabetes on Twitter.
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: Diabetic coma and nerve damage| Publication types | A person unconscious from hypoglycemia is usually pale, has a rapid heart beat, and is soaked in sweat: all signs of the adrenaline response to hypoglycemia. The individual is not usually dehydrated and breathing is normal or shallow. Their blood sugar level, measured by a glucose meter or laboratory measurement at the time of discovery, is usually low but not always severely, and in some cases may have already risen from the nadir that triggered the unconsciousness. Unconsciousness due to hypoglycemia is treated by raising the blood glucose with intravenous glucose or injected glucagon. Diabetic ketoacidosis DKA , most typically seen in those with type 1 diabetes, is triggered by the build-up of chemicals called ketones. These are strongly acidic and a build-up can cause the blood to become acidic. If it progresses and worsens without treatment it can eventually cause unconsciousness, from a combination of a very high blood sugar level, dehydration and shock , and exhaustion. Coma only occurs at an advanced stage, usually after 36 hours or more of worsening vomiting and hyperventilation. In the early to middle stages of ketoacidosis, patients are typically flushed and breathing rapidly and deeply, but visible dehydration, pale appearance from diminished perfusion, shallower breathing, and a fast heart rate are often present when coma is reached. However these features are variable and not always as described. If the patient is known to have diabetes, the diagnosis of diabetic ketoacidosis is usually suspected from the appearance and a history of 1—2 days of vomiting. The diagnosis is confirmed when the usual blood chemistries in the emergency department reveal a high blood sugar level and severe metabolic acidosis. Treatment of diabetic ketoacidosis consists of isotonic fluids to rapidly stabilize the circulation, continued intravenous saline with potassium and other electrolytes to replace deficits, insulin to reverse the ketoacidosis, and careful monitoring for complications. Nonketotic hyperosmolar coma usually develops more insidiously than diabetic ketoacidosis because the principal symptom is lethargy progressing to obtundation , rather than vomiting and an obvious illness. Extremely high blood sugar levels are accompanied by dehydration due to inadequate fluid intake. Coma occurs most often in patients who have type 2 or steroid diabetes and have an impaired ability to recognize thirst and drink. It is classically a nursing home condition but can occur in all ages. The treatment consists of insulin and gradual rehydration with intravenous fluids. Diabetic coma was a more significant diagnostic problem before the late s, when glucose meters and rapid blood chemistry analyzers were not available in all hospitals. In modern medical practice, it rarely takes more than a few questions, a quick look, and a glucose meter to determine the cause of unconsciousness in a patient with diabetes. Laboratory confirmation can usually be obtained in half an hour or less. Other conditions that can cause unconsciousness in a person with diabetes are stroke, uremic encephalopathy, alcohol, drug overdose, head injury, or seizure. Most patients do not reach the point of unconsciousness or coma in cases of diabetic hypoglycemia, diabetic ketoacidosis, or severe hyperosmolarity before a family member or caretaker seeks medical help. Treatment depends upon the underlying cause: [7]. Contents move to sidebar hide. Article Talk. Read Edit View history. Tools Tools. What links here Related changes Upload file Special pages Permanent link Page information Cite this page Get shortened URL Download QR code Wikidata item. Download as PDF Printable version. Medical condition. This article needs additional citations for verification. Please help improve this article by adding citations to reliable sources. Unsourced material may be challenged and removed. Find sources: "Diabetic coma" — news · newspapers · books · scholar · JSTOR June Learn how and when to remove this template message. International Diabetes Federation. Diabetic Ketoacidosis is common amongst people with type 1 diabetes and is triggered by the build-up of ketones. Ketones build up when sugar levels are too low and the body begins to burn fat for energy. Diabetic hyperosmolar occurs when your blood sugar is dangerously high, causing your blood to get thick and syrupy. Your body will try to get rid of the excess sugar by passing it in urine. This will cause frequent urination that can lead to dehydration. If you are experiencing these symptoms check your blood glucose levels right away and seek immediate medical attention. The best way to avoid diabetic comas is through prevention and managing your diabetes. You can manage your diabetes by checking your blood sugar regularly, exercising, sticking to your recommended diet, staying hydrated and taking the proper doses of medication. For more information on diabetes management and early prevention Jamaica Hospital offers a free diabetes prevention program, to sign up please call All content of this newsletter is intended for general information purposes only and is not intended or implied to be a substitute for professional medical advice, diagnosis or treatment. |
| Actions for this page | We nerrve products we think are useful for our readers. News Network. Damagw blood glucose hypoglycemia. Nerve Diabetic coma and nerve damage is one possible complication from having high blood sugar levels for a long time. Anyone who has diabetes is at risk of a diabetic coma, but the following factors can increase the risk: Insulin delivery problems. Accessed July 11, |
| What Happens During A Diabetic Coma | A diabetic coma is unlikely as long as you take your medications as prescribed and monitor your blood glucose levels routinely. It can occur in people with type 1 or type 2 diabetes. Diabetic ketoacidosis DKA occurs when your body lacks insulin and uses fat instead of glucose for energy. Having frequent episodes of hyperglycemia high blood sugar can stress the brain. Jun 28, Written By Kristeen Cherney, PhD. Coma and other neurologic abnormalities are present in patients with either diabetic ketoacidosis DKA or nonketotic coma NKC , and the cause of such phenomena are not known. |
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